Volume 11 Issue 2
Mar.  2020
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Yang Jian, Xie Yan, Tian Dazhi, et al. Research progress on molecular mechanism of hepatitis B virus reactivation after liver transplantation[J]. ORGAN TRANSPLANTATION, 2020, 11(2): 298-303. doi: 10.3969/j.issn.1674-7445.2020.02.019
Citation: Yang Jian, Xie Yan, Tian Dazhi, et al. Research progress on molecular mechanism of hepatitis B virus reactivation after liver transplantation[J]. ORGAN TRANSPLANTATION, 2020, 11(2): 298-303. doi: 10.3969/j.issn.1674-7445.2020.02.019

Research progress on molecular mechanism of hepatitis B virus reactivation after liver transplantation

doi: 10.3969/j.issn.1674-7445.2020.02.019
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  • Corresponding author: Jiang Wentao, Email: 447660280@qq.com
  • Received Date: 2019-12-20
    Available Online: 2021-01-19
  • Publish Date: 2020-03-15
  • Liver transplantation is the most effective method for hepatitis B-related liver failure, liver cirrhosis and hepatocellular carcinoma. However, the reactivation of hepatitis B virus (HBV) after liver transplantation is not conducive to the recovery of liver function and leads to poor clinical prognosis. The prevention and treatment of HBV reactivation is currently the focus of research by physicians and surgeons. The current viral suppression strategies can not completely eradicate HBV nor completely prevent the recurrence of HBV infection in the future. This article aims to explore the molecular mechanism of HBV reactivation after liver transplantation, in order to more effectively prevent the recurrence of hepatitis B after liver transplantation.

     

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